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Rodrigues, Richard R. Greer, Renee L. Dong, Xiaoxi DSouza, Karen N. Gurung, Manoj Wu, Jia Y. Morgun, Andrey and Shulzhenko, Natalia 2017. Antibiotic-Induced Alterations in Gut Microbiota Are Associated with Changes in Glucose Metabolism in Healthy Mice. Frontiers in Microbiology, Vol. 8, Issue. ,
  • CrossRef
  • Google Scholar
Laessle, Claudia Nenova, Gergana Marjanovic, Goran Seifert, Gabriel Kousoulas, Lampros Jaenigen, Bernd Fichtner-Feigl, Stefan and Fink, Jodok Matthias 2018. Duodenal Exclusion but Not Sleeve Gastrectomy Preserves Insulin Secretion, Making It the More Effective Metabolic Procedure. Obesity Surgery, Vol. 28, Issue. 5, p. 1408.
  • CrossRef
  • Google Scholar
Laferrère, Blandine and Pattou, François 2018. Weight-Independent Mechanisms of Glucose Control After Roux-en-Y Gastric Bypass. Frontiers in Endocrinology, Vol. 9, Issue. ,
  • CrossRef
  • Google Scholar
Hegyi, Peter Maléth, Jozsef Walters, Julian R. Hofmann, Alan F. and Keely, Stephen J. 2018. Guts and Gall: Bile Acids in Regulation of Intestinal Epithelial Function in Health and Disease. Physiological Reviews, Vol. 98, Issue. 4, p. 1983.
  • CrossRef
  • Google Scholar
Rajagopal, Senthilkumar and Ponnusamy, Murugavel 2018. Metabotropic GPCRs: TGR5 and P2Y Receptors in Health and Diseases. p. 19.
  • CrossRef
  • Google Scholar
Wang, Xuan Li, Shuo Chen, Man Liu, Jing Dong, Ruirui Wang, Huamin and Zhu, Shigong 2018. Activation of the Nuclear Receptor Fxr Improves Intestinal Cell Tolerance to Ischemia–Reperfusion Injury. Shock, Vol. 50, Issue. 3, p. 316.
  • CrossRef
  • Google Scholar
Zhang, Fan Yuan, Wenzhen Wei, Yuhui Zhang, Dongmei Duan, Yingting Li, Boxia Wang, Xiaohui Xi, Lili Zhou, Yan and Wu, Xinan 2019. The alterations of bile acids in rats with high-fat diet/streptozotocin-induced type 2 diabetes and their negative effects on glucose metabolism. Life Sciences, Vol. 229, Issue. , p. 80.
  • CrossRef
  • Google Scholar
Cerdó, Tomás García-Santos, José G. Bermúdez, Mercedes and Campoy, Cristina 2019. The Role of Probiotics and Prebiotics in the Prevention and Treatment of Obesity. Nutrients, Vol. 11, Issue. 3, p. 635.
  • CrossRef
  • Google Scholar
Qi, Li Tian, Yu and Chen, Yongsheng 2019. Gall bladder: The metabolic orchestrator. Diabetes/Metabolism Research and Reviews, Vol. 35, Issue. 5,
  • CrossRef
  • Google Scholar
de Boer, Jan Freark Verkade, Esther Mulder, Niels L. de Vries, Hilde D. Huijkman, Nicolette Koehorst, Martijn Boer, Theo Wolters, Justina C. Bloks, Vincent W. van de Sluis, Bart and Kuipers, Folkert 2020. A human-like bile acid pool induced by deletion of hepatic Cyp2c70 modulates effects of FXR activation in mice. Journal of Lipid Research, Vol. 61, Issue. 3, p. 291.
  • CrossRef
  • Google Scholar
Mohr, Alex E. Gumpricht, Eric Sears, Dorothy D. and Sweazea, Karen L. 2021. Recent advances and health implications of dietary fasting regimens on the gut microbiome. American Journal of Physiology-Gastrointestinal and Liver Physiology, Vol. 320, Issue. 5, p. G847.
  • CrossRef
  • Google Scholar
Sun, Runbin Kong, Bo Yang, Na Cao, Bei Feng, Dong Yu, Xiaoyi Ge, Chun Feng, Siqi Fei, Fei Huang, Jingqiu Lu, Zhenyao Xie, Yuan Yang, Chung S. Guo, Grace L. Wang, Guangji and Aa, Jiye 2021. The Hypoglycemic Effect of Berberine and Berberrubine Involves Modulation of Intestinal Farnesoid X Receptor Signaling Pathway and Inhibition of Hepatic Gluconeogenesis. Drug Metabolism and Disposition, Vol. 49, Issue. 3, p. 276.
  • CrossRef
  • Google Scholar
Engin, Atilla 2021. Protein Kinase-mediated Decisions Between Life and Death. Vol. 1275, Issue. , p. 229.
  • CrossRef
  • Google Scholar
Durník, Robin Šindlerová, Lenka Babica, Pavel and Jurček, Ondřej 2022. Bile Acids Transporters of Enterohepatic Circulation for Targeted Drug Delivery. Molecules, Vol. 27, Issue. 9, p. 2961.
  • CrossRef
  • Google Scholar
Yan, Meng Hou, Li Cai, Yaoyao Wang, Hanfei Ma, Yujun Geng, Qiming Jiang, Weiwei and Tang, Weibing 2022. Effects of Intestinal FXR-Related Molecules on Intestinal Mucosal Barriers in Biliary Tract Obstruction. Frontiers in Pharmacology, Vol. 13, Issue. ,
  • CrossRef
  • Google Scholar
Gao, Ruolin Meng, Xiangjing Xue, Yili Mao, Min Liu, Yaru Tian, Xuewen Sui, Bo Li, Xun and Zhang, Pengyi 2022. Bile acids-gut microbiota crosstalk contributes to the improvement of type 2 diabetes mellitus. Frontiers in Pharmacology, Vol. 13, Issue. ,
  • CrossRef
  • Google Scholar
Lawal, Yakubu 2022. Gut dysbiosis. Journal of Current Research in Scientific Medicine, Vol. 8, Issue. 1, p. 12.
  • CrossRef
  • Google Scholar
Mooranian, Armin Chester, Jacqueline Johnston, Edan Ionescu, Corina Mihaela Walker, Daniel Jones, Melissa Wagle, Susbin Raj Kovacevic, Bozica Foster, Thomas Mikov, Momir and Al-Salami, Hani 2022. Reduced Cytokine Tumour Necrosis Factor by Pharmacological Intervention in a Preclinical Study. Biomolecules, Vol. 12, Issue. 7, p. 877.
  • CrossRef
  • Google Scholar
Wachsmuth, Hallie R. Weninger, Savanna N. and Duca, Frank A. 2022. Role of the gut–brain axis in energy and glucose metabolism. Experimental & Molecular Medicine, Vol. 54, Issue. 4, p. 377.
  • CrossRef
  • Google Scholar
Esan, Oluwayemisi Viljoen, Adie and Wierzbicki, Anthony S. 2022. Colesevelam – a bile acid sequestrant for treating hypercholesterolemia and improving hyperglycemia. Expert Opinion on Pharmacotherapy, Vol. 23, Issue. 12, p. 1363.
  • CrossRef
  • Google Scholar
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Intestinal bile acid receptors are key regulators of glucose homeostasis
  • Volume 76, Issue 3
  • Mohamed-Sami Trabelsi (a1), Sophie Lestavel (a2), Bart Staels (a2) and Xavier Collet (a1)
  • DOI: https://doi.org/10.1017/S0029665116002834
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Intestinal bile acid receptors are key regulators of glucose homeostasis
  • Volume 76, Issue 3
  • Mohamed-Sami Trabelsi (a1), Sophie Lestavel (a2), Bart Staels (a2) and Xavier Collet (a1)
  • DOI: https://doi.org/10.1017/S0029665116002834
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Intestinal bile acid receptors are key regulators of glucose homeostasis
  • Volume 76, Issue 3
  • Mohamed-Sami Trabelsi (a1), Sophie Lestavel (a2), Bart Staels (a2) and Xavier Collet (a1)
  • DOI: https://doi.org/10.1017/S0029665116002834
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